Browsing publications by Dr Marzena Ciechomska

Newcastle AuthorsTitleYear
Dr Marzena Ciechomska
Dr Caroline Wilson
Achilleas FLOUDAS
Professor Drew Rowan
Professor John Robinson
et al.
Antigen-specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD4+ T-cell activation2014
Dr Steven O'Reilly
Rachel Cant
Dr Marzena Ciechomska
Dr Fiona Oakley
Professor Sophie Hambleton
et al.
Serum Amyloid A (SAA) induces IL-6 in dermal fibroblasts via TLR2, IRAK4 and NF-κB2014
Dr Steven O'Reilly
Rachel Cant
Dr Marzena Ciechomska
Professor Jaap Van Laar
Interleukin-6: a new therapeutic target in systemic sclerosis?2013
Dr Marzena Ciechomska
Rachel Cant
James Finnigan
Professor Jaap Van Laar
Dr Steven O'Reilly
et al.
Role of toll-like receptors in systemic sclerosis2013
Dr Marzena Ciechomska
Christiaan Huigens
Tess Stanly
Dr Andreas Gessner
Professor Sophie Hambleton
et al.
Toll-like receptor-mediated, enhanced production of profibrotic TIMP-1 in monocytes from patients with systemic sclerosis: role of serum factors2013
Dr Marzena Ciechomska
Christiaan Huigens
Dr Andreas Gessner
Dr Steven O'Reilly
Professor Jaap Van Laar
et al.
A novel role of monocytes in the induction of profibrotic tissue-inhibitor of metalloproteinase-1 mediated by toll-like receptors stimulation in SSC2012
Dr Steven O'Reilly
Dr Marzena Ciechomska
Professor Jaap Van Laar
Interleukin-6, its role in fibrosing conditions2012
Dr Marzena Ciechomska
Dr Andreas Gessner
Dr Steven O'Reilly
Professor Jaap Van Laar
Toll-like receptor-mediated expression of profibrotic tissue-inhibitor of metalloproteinase-1 by circulating monocytes in Systemic sclerosis2012
Dr Marzena Ciechomska
Dr Caroline Wilson
Professor Drew Rowan
Professor John Robinson
Dr Andrew Knight
et al.
Antigen-specific B cells can acquire and present the rheumatoid arthritis candidate autoantigen aggrecan from a non-internalisable surface2011
Dr Marzena Ciechomska
Professor Thomas Lennard
Professor John Kirby
Dr Andrew Knight
B lymphocytes acquire and present intracellular antigens that have relocated to the surface of apoptotic target cells2011
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