Browsing publications by Professor Michael Briggs

Newcastle AuthorsTitleYearFull text
Dr Katarzyna Pirog
Professor Michael Briggs
Cartilage-Specific Ablation of XBP1 Signaling in Mouse Results in a Chondrodysplasia Characterized by Reduced Chondrocyte Proliferation and Delayed Cartilage Maturation and Mineralization2015
Professor Michael Briggs
Increased Classical Endoplasmic Reticulum Stress Is Sufficient to Reduce Chondrocyte Proliferation Rate in the Growth Plate and Decrease Bone Growth2015
Professor Michael Briggs
Dr Peter Bell
Dr Katarzyna Pirog
The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review)2015
Dr Katarzyna Pirog
Stacy Young
Dr Peter Bell
Professor Michael Briggs
Abnormal Chondrocyte Apoptosis in the Cartilage Growth Plate is Influenced by Genetic Background and Deletion of CHOP in a Targeted Mouse Model of Pseudoachondroplasia2014
Professor Michael Briggs
Dr Peter Bell
Genotype to phenotype correlations in cartilage oligomeric matrix protein associated chondrodysplasias2014
Dr Benedetta Gualeni
Dr Peter Bell
Professor Michael Briggs
A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growth2013
Dr Peter Bell
Professor Michael Briggs
Analysis of the cartilage proteome from three different mouse models of genetic skeletal diseases reveals common and discrete disease signatures2013
Dr Peter Bell
Professor Michael Briggs
Armet/Manf and Creld2 are components of a specialized ER stress response provoked by inappropriate formation of disulphide bonds: implications for genetic skeletal diseases2013
Professor Michael Briggs
Chondrocyte ER stress as a pathogenic factor in osteoarthritis2013
Dr Katarzyna Pirog
Professor Michael Briggs
Mild myopathy is associated with COMP but not MATN3 mutations in mouse models of genetic skeletal diseases2013
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