Browsing publications by Professor Michael Briggs

Newcastle AuthorsTitleYear
Dr Katarzyna Pirog
Stacy Young
Dr Peter Bell
Professor Michael Briggs
Abnormal Chondrocyte Apoptosis in the Cartilage Growth Plate is Influenced by Genetic Background and Deletion of CHOP in a Targeted Mouse Model of Pseudoachondroplasia2014
Professor Michael Briggs
Dr Peter Bell
Genotype to phenotype correlations in cartilage oligomeric matrix protein associated chondrodysplasias2014
Dr Benedetta Gualeni
Dr Peter Bell
Professor Michael Briggs
A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growth2013
Dr Peter Bell
Professor Michael Briggs
Analysis of the cartilage proteome from three different mouse models of genetic skeletal diseases reveals common and discrete disease signatures2013
Dr Peter Bell
Professor Michael Briggs
Armet/Manf and Creld2 are components of a specialized ER stress response provoked by inappropriate formation of disulphide bonds: implications for genetic skeletal diseases2013
Professor Michael Briggs
Chondrocyte ER stress as a pathogenic factor in osteoarthritis2013
Dr Katarzyna Pirog
Professor Michael Briggs
Mild myopathy is associated with COMP but not MATN3 mutations in mouse models of genetic skeletal diseases2013
Dr Katarzyna Pirog
Professor Michael Briggs
Mutant chondrocytes are characterised by altered cell shape and changes to the organisation of primary cilia in a genetic mouse model of mild pseudoachondroplasia2013
Dr Katarzyna Pirog
Professor Michael Briggs
A novel form of chondrocyte stress is triggered by a COMP mutation causing pseudoachondroplasia2012
Dr Peter Bell
Dr Katarzyna Pirog
Professor Michael Briggs
Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation2012
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