Browsing publications by Professor Simon Bailey

Newcastle AuthorsTitleYearFull text
Dr Rebecca Hill
Dr Janet Lindsey
Dr Ed Schwalbe
Dr Karen Barker
Dr Daniel Williamson
et al.
Combined MYC and P53 Defects Emerge at Medulloblastoma Relapse and Define Rapidly Progressive, Therapeutically Targetable Disease2015
Dr Janet Lindsey
Dr Ed Schwalbe
Sandeep Potluri
Professor Simon Bailey
Dr Daniel Williamson
et al.
TERT promoter mutation and aberrant hypermethylation are associated with elevated expression in medulloblastoma and characterise the majority of non-infant SHH subgroup tumours2014
Dr Martina Finetti
Matthew Selby
Dr Stephen Crosier
Ben Skalkoyannis
Professor Simon Bailey
et al.
Characterising critical pathways in ATRT tumourigenesis: a genome-wide analysis of primary tumours and functional genomic models2014
Professor Simon Bailey
Dr Janet Lindsey
Professor Steven Clifford
Cytogenetic Prognostication Within Medulloblastoma Subgroups2014
Dr Nicholas Bown
Dr Sujith Samarasinghe
Professor Simon Bailey
Dr Roderick Skinner
FLT3 internal tandem duplication and NPM1 mutations in paediatric AML: a single centre experience2014
Professor Simon Bailey
Professor David Ellison
Professor Steven Clifford
Improved health-related quality of life outcomes associated with SHH subgroup medulloblastoma in SIOP-UKCCSG PNET3 trial survivors2014
Professor Simon Bailey
Management of Children with Wilms Tumor in Africa and Europe; Thoughts About Costs, Priorities and Collaboration2014
Dr Martina Finetti
Ben Skalkoyannis
Matthew Selby
Dr Stephen Crosier
Professor Simon Bailey
et al.
Mechanism of tumourigenesis caused by loss of SMARCB1 in malignant rhabdoid tumors2014
Professor Simon Bailey
Professor Steven Clifford
Dr Daniel Williamson
Medulloblastoma Down Under 2013: a report from the third annual meeting of the International Medulloblastoma Working Group2014
Dr Rebecca Hill
Dr Janet Lindsey
Dr Ed Schwalbe
Dr Daniel Williamson
Dr Sarra Ryan
et al.
MYC and TP53 defects emerge and interact at medulloblastoma relapse, define rapidly progressive disease and can be targeted therapeutically2014
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