Browsing publications by Dr David Sheridan

Newcastle AuthorsTitleYear
Professor Margaret Bassendine
Dr David Sheridan
Jaime Jonsson
CCR5-Δ32 genotype does not improve predictive value of IL28B polymorphisms for treatment response in chronic HCV infection2013
Dr David Sheridan
Dr R Neely
Professor Margaret Bassendine
Hepatitis C virus and lipids in the era of direct acting antivirals (DAAs)2013
Professor Margaret Bassendine
Dr David Sheridan
Dr Simon Bridge
Dr Daniel Felmlee
Dr R Neely
et al.
Lipids and HCV2013
Dr David Sheridan
Dr Simon Bridge
Dr Daniel Felmlee
Fiona Fenwick
Professor Margaret Bassendine
et al.
Omega-3 Fatty Acids and/or Fluvastatin in Hepatitis C Prior Non-Responders to Combination Anti-Viral Therapy - A Pilot Randomised Clinical Trial2013
Dr David Sheridan
Dr Simon Bridge
Dr Daniel Felmlee
Professor Geoff Toms
Dr R Neely
et al.
Apolipoprotein-E and hepatitis C lipoviral particles in genotype 1 infection: Evidence for an association with interferon sensitivity2012
Professor Margaret Bassendine
Dr David Sheridan
IL28B, HLA-C, and KIR Variants Additively Predict Response to Therapy in Chronic Hepatitis C Virus Infection in a European Cohort: A Cross-Sectional Study2011
Dr Simon Bridge
Dr David Sheridan
Dr Daniel Felmlee
Professor Geoff Toms
Professor Margaret Bassendine
et al.
Apolipoprotein E and low-density, apolipoprotein B associated lipoviral particles in chronic Hepatitis C infection: Evidence for genotype-specific modulation of lipid pathways2011
Professor Margaret Bassendine
Dr David Sheridan
Dr Daniel Felmlee
Dr Simon Bridge
Professor Geoff Toms
et al.
HCV and the hepatic lipid pathway as a potential treatment target2011
Dr Daniel Felmlee
Dr David Sheridan
Dr Simon Bridge
Professor Geoff Toms
Professor Margaret Bassendine
et al.
Use of intralipid infusion to analyse apolipoprotein B (APOB) and HCV RNA kinetics in chronic infection2011
Dr David Sheridan
Professor Kim Bartlett
Dr Daniel Felmlee
Dr Simon Bridge
Professor Geoff Toms
et al.
Hypocholesterolaemia in chronic hepatitis C genotype 3 infection is due to both reduced cholesterol production and increased LDL clearance2010
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