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Ventriculoatrial synchrony induced heart failure

Lookup NU author(s): Dr Djordje Jakovljevic

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Abstract

© Acta Clinica Belgica 2018 Objective and importance: Endless loop tachycardia or pacemaker-mediated tachycardia, and atrioventricular desynchronization arrhythmia or repetitive non-reentrant ventriculoatrial synchrony (RNRVAS) are two forms of reverse impulse conduction – ventriculoatrial (VA) synchrony. Although VA synchrony can theoretically cause aggravation of heart failure, clinical cases describing severe consequential heart failure are lacking. Clinical presentation and intervention: We describe a case of a 60-year-old patient who underwent primary percutaneous coronary intervention and mitral valve surgery. Implantation of a two-chamber pacemaker was also performed during the same hospitalization due to development of third-degree atrioventricular block. Ten months later, he presented with a severe form of heart failure with a significant reduction of left ventricular ejection fraction (LVEF). The atrial lead was displaced and VA synchrony was registered (RNRVAS-like condition). The pacemaker was reprogrammed and VA synchrony induced heart failure was successfully resolved. Echocardiographic follow-up showed improvement in LVEF. Conclusion: Ventriculoatrial conduction can be present even when the patient has a complete atrioventricular block. Atrial lead displacement and consequently loss of atrial capture with preserved sensing can be a predisposing factor for initiation of ventriculoatrial synchrony. Permanent ventriculoatrial synchrony may provoke aggravation of heart failure.


Publication metadata

Author(s): Andric S, Tesic D, Somer D, Srdanovic I, Miljkovic T, Jakovljevic DG, Velicki L

Publication type: Article

Publication status: Published

Journal: Acta Clinica Belgica: International Journal of Clinical and Laboratory Medicine

Year: 2018

Volume: 73

Issue: 6

Pages: 439-443

Online publication date: 21/01/2018

Acceptance date: 02/04/2016

ISSN (print): 1784-3286

ISSN (electronic): 2295-3337

Publisher: Taylor and Francis Ltd

URL: https://doi.org/10.1080/17843286.2018.1443002

DOI: 10.1080/17843286.2018.1443002


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