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Browsing publications by Dr Laura Greaves

Newcastle AuthorsTitleYearFull text
Dr Dave Houghton
Dr Christopher Stewart
Dr Craig Stamp
Professor Anil Wipat
Professor Michael Trenell
et al.
Age-related mitochondrial dysfunction influences the mouse intestinal microbiome composition2018
Dr Dave Houghton
Dr Christopher Stewart
Dr Craig Stamp
Professor Anil Wipat
Professor Michael Trenell
et al.
Impact of Age-Related Mitochondrial Dysfunction and Exercise on Intestinal Microbiota Composition2017
Alex Bury
Dr Angela Pyle
Dr Joanna Elson
Dr Laura Greaves
Dr Christopher Morris
et al.
Mitochondrial DNA changes in pedunculopontine cholinergic neurons in Parkinson disease2017
Dr Mohammad Moad
Laura Wilson
Rob Pickard
Professor Doug Turnbull
Professor Robert Taylor
et al.
Multipotent Basal Stem Cells, Maintained in Localized Proximal Niches, Support Directed Long-Ranging Epithelial Flows in Human Prostates2017
Holly Baines
Dr Craig Stamp
Dr Laura Greaves
A Phenotype-Driven Approach to Generate Mouse Models with Pathogenic mtDNA Mutations Causing Mitochondrial Disease2016
Ashwin Sachdeva
Dr Amira El-Sherif
Professor Doug Turnbull
Dr Laura Greaves
Dr Rakesh Heer
et al.
Development of prostate intra-epithelial neoplasia in an aging series of PolgA mutator mice suggests a role for mitochondrial DNA mutations in prostate carcinogenesis2016
Dr Ellen Tullo
Dr Laura Greaves
Dr Luisa Wakeling
Involving older people in the design, development, and delivery of an innovative module on aging for undergraduate students2016
Dr Clara Correia Melo
Francisco Marques
Rhys Anderson
Dr Graeme Hewitt
Dr Bernadette Carroll
et al.
Mitochondria are required for pro-ageing features of the senescent phenotype2016
Dr Philip Dobson
Dr Mariana Rocha
Dr John Grady
Alexia Chrysostomou
Sharon Watson
et al.
Unique quadruple immunofluorescence assay demonstrates mitochondrial respiratory chain dysfunction in osteoblasts of aged and PolgA -/- mice2016
Dr Grainne Gorman
Dr Hue Hornig - Do
Dr Helen Tuppen
Dr Laura Greaves
Dr Langping He
et al.
Novel MTND1 mutations cause isolated exercise intolerance, complex I deficiency and increased assembly factor expression2015
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